Glocom Pathology: What to Know

The article was consulted professionally by Specialist Doctor II Nguyen Thai Hung – Department of Medical Examination and Internal Medicine – Share99 Da Nang International Hospital.

Glaucoma is a medical condition caused by a number of mechanisms that cause increased pressure in the eyes. If not treated properly, then when the glaucoma increases, it causes damage to the visual nerves. Lesions caused by glaucoma pathology that adversely affect the visual nerve are i.e.

1.1. mechanics

Glaucoma is a factor that is considered the cause of a chain of disease changes in the cellular level of the neuron market. The optics consist of axial filaments starting from the location of VM ganglia (unmalined ganglia) and then out of the eyeball (myelinized) through the corneal scytheum leaf, a collagen-like structure.

• Pressure on unmaxed lymphocytes

Increased glaucoma acts directly on myelinized lymphocytes, including the axial guided activity of the axial, thereby killing the ganglia due to the lack of neuron nutritional factors derived from the brain. Prolonged reduction of neurological perfusion causes tissue anemia leading to the accumulation of retinobular radicals, resulting in damaged and undernourished lymphocytes.

In glaucoma, glaucoma is the cause of the activation of spores at the end of the neuron ( buffer neurons, a star cells). Activated acythic cells synthesize molecules that cause degeneration and restructuring of the external organization while having a mechanical and biological impact on the head of the nerve and VM ganglia.

1.2. Immunity

In terms of immunity, it has been suggested that the compression of axial soy sauce cells in the corneal scystinal leaves can prevent the conduction of signals from axial fibers to the ganglia stem leading to programmable death. This phenomenon involves the activation of ENZYMES that digest DNA in the ion, and the realization of cell structure framing components by the cells next to it.

When there are syscs caused by glaucoma, the interaction between programmable oncology signals and neuronutrient factors is what determines the existence of VM ganglion cells.

1.3. blood vessel

Many studies have mentioned the link between the progress of Glaucoma to migraine and other vascular diseases such as hypertension, diabetes mellitus. Using flow survey techniques such as Pulsatile Ocular Blood Flowmeter, Scanning Laser Doppler Flowmetry, Color Doppler Imaging, the authors detected a decrease in perfuscency to various locations of the eyeball including the nerve, melanoma, retina and posterior vascular system in cases of Glaucoma. In addition, studies have shown disorders in the ability to self-regulate blood flow (according to the mechanisms of conversion, hormones, nerves) when there is a decrease in perfusation of the area of the nerve market.

2.1 Mechanism of pathogenescies in primitive closed-angle glaucoma

On the eyes with a larger-than-usual glass structure, or the position of the glass body protruding first than the average person, then the front of the glass body will be close to the back of the iris causing congestion of the uterus. The fluid does not escape to the front of the room, will be stagnant in the back of the room and the posterity pressure increases, the iris is pushed out before the pressure on the cornea raft area causing symptoms of closing the corner. Fluid retention in the eyeball is the cause of glaucoma.

Congestion in front of the raft area – cornea (by the mechanism of closing the angle)

In the eyes with a small corneal structure, shallow room money, a narrow pre-room corner, when the homage dilates the thick iris legs attached to the back of the cornea, the pre-room corner is closed. The fluid does not escape through the raft area into the vein system, which causes fluid to stagn in the eyeball causing glaucoma.

Closed-angle glaucoma can occur without congestion of the homage. In some cases, the iris surface is flat, the room rate in the center seems deeper than usual. This phenomenon is caused by deformity of the iris, the lack of congestion of the homage. After the hosimology stretches the perimeter of the iris and seals into the raft area causing glaucoma to close the corner.

2.2. Pathogenesa in primitive open-angle glaucoma

The pathogenesa of primitive open-angle glaucoma is unknown. Glaucoma usually corresponds to nerve vision damage caused by circulating disorders that supply blood to the eye plate and glaucoma caused by disorders of the circulation of hydrolysers in the raft area.

Glaucoma is a rather dangerous disease and causes blindness. However, you can completely avoid this risk if you detect the disease early. Share99 International Health Hub is currently providing a Glaucoma Package that will perform comprehensive examinations and prescribe surgery (if any) to customers with symptoms of Glaucoma.

Specialist 2 Nguyen Thai Hung has 13 years of experience as a doctor treating eye diseases at C Health Hub Da Nang and Ngu Hanh Son General Health Hub – Da Nang. Currently, He is an Eye Doctor at The Interdisciplinary Clinic of Medical Examination and Internal Medicine at Share99 Da Nang International Health Hub.

For more information about the Glaucoma package, please contact: 02033 828 188 or make an reservation HERE

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