In-infant diaphragm: What to know

In-infant diaphragm is one of the common acute respiratory distress diseases, common in premature babies. Inner membrane disease is caused by incomplete development of the respiratory tract and a deficiency of surfactant, an active substance on the inner side of the alveolar sculus.

1. What is in-infant diaphragm?

In-membrane disease is one of the most common causes of acute respiratory failure in babies, especially in the group of premature babies. It is also the most fatal pathology in premature babies.

Diaphragm in premature babies occurs because the lungs of the child are not really mature. The surfactant deficiency causes the alveolar vesiclia inside the child's lungs to collapse, plasma from the capillaries overflows the alveoli and interferes with breathing. Children with diaphragm usually present with severe acute respiratory failure after birth for a few minutes to several hours, eliminating other causes associated with infection, inhalation of amniotic fluid stools.

Intratermeal disease in premature babies is a disease that is effectively pre-birth if well managed pregnancy. Pregnant women with a history of premature birth,or other abnormalities that increase the likelihood of premature labor such as a short cervical or infection, should be monitored more closely for timely treatment and prevention of respiratory failure for the child.

fatal anaphylactic shock for children

In-membrane disease is one of the leading causes of death in babies

2. Causes of pericarditis in newborns

Surfactant deficiency and an underdest developed respiratory system are the direct causes of membrane disease in premature babies. Surfactant is the active substance located inside the alveoli, which is responsible for maintaining the surface tension of the alveolar vesicpas, keeping them from collapsing throughout the respiratory process. In a healthy fetus, type II alveolar cells begin to secrete surfactant from the 20th week of gestation and the alveolar system develops completely at the 32nd week of gestation.

Premature babies with inner membrane disease face a decrease in the area of the alveolar gas exchange surface due to pulmonary collapse, hypoxia to feed the body leading to blood acidification, acute and chronic damage to the lung cuticle. Lesions in the lungs continue to hinder the production of surfactant, increasing the edema of the alveolar sclelar sclealas due to fluid retention and deposition of fibrin fibers.

In-infant diaphragm is divided into two main groups: Primary in-domestic membrane disease that occurs in premature babies, and second-in-the-middle membrane disease that appears in full-month and nearly full-month babies. Thus, the disease of the membranes in the membrane does not occur only with the group of premature babies. The incidence of membrane disease in full-month babies is about 5% and is rare in pregnancies over 38 weeks. Some of the risk factors that increase the risk of appearance of membrane disease in newborns are:

  • Increased glucose levels in the mother's blood.
  • Mother with gestational diabetes mellitus.
  • High levels of insulin in the mother's blood.
  • Child inhaling stools
  • Inflammatory conditions in lung mesenteritis
  • Cesarean section

When to screen for gestational diabetes with high-risk people

Mother with gestational diabetes increases baby's risk of disease

3. Signs of recognition of membrane disease in newborns

In-infant diaphragm is characterized by acute respiratory failure that occurs early after birth. Symptoms usually develop about the first 6 hours after birth, some mild cases may be after a few days. Respiratory failure appears suddenly with such manifestations as:

  • Cyanosis of the skin and mucous membranes, progressing gradually over time.
  • The frequency of breathing increases rapidly. In extremely premature babies, the frequency of breathing may be slow.
  • Fast circuit
  • Traces of use of secondary respiratory muscles: nasal bulging, thymus contraction
  • The lungs hear decreased ventilation. The lung area reduces ventilation spreading from the resident to the entire lungs.
  • If not detected and treated in a timely manner, the child with an in clear membrane disease will fall into a severe state with symptoms of wrestling, yawning, low frequency of breathing, the appearance of apnea and death.

Respiratory failure

Acute respiratory distress occurs about the first 6 hours after birth

4. Methods of diagnosing diaphragm in newborns

Diagnosis of in-domestic membrane disease in newborns requires coordination between the exploitation of risk factors of the disease, early symptoms of disease detection and additional subclinical tests. Diaphragm usually occurs with high frequency in premature babies, asphyxiated by inhalation of stools and mothers with diabetes mellitus. Clinical manifestations are mainly severe respiratory distress, which progresses to deteriorate over time. When a child with an insural membrane disease is suspected, subclinical means in place to assist in clarifying commonly used diagnosis include:

  • Pulmonary X-ray:Internal membrane disease in newborns with translucent nodular lesions scattered on the sides of the lungs accompanied by images of gas retention lesions in the bronchi. In severe cases, the interstitial tissue in the lungs is edema that creates a mesh blurred image or blurs all two scraps when there is a collapse of the lungs.
  • Arterial blood gas: Helps identify acute respiratory failure and blood acidification. Acidosis in the blood is a bad sign of a child's reed.
  • Bio-testing of amniotic fluid, bronchial or gastric secrety determines premature condition in a child.
  • Anatomy: Taking tissue from the lungs as an anatomy to accurately diagnose the disease is only carried out resuscitation on the corpse. Lung lesions are characterized by alveolar deflated, alveoli contain many inflammatory cells and fear of fibrin, conjuncular tissue conjuncence.

products

Specimen testing to make an accurate diagnosis

5. Treatments for pericarditis in newborns

The effectiveness of the treatment of diaphragm in babies depends on when the disease is detected for treatment. Children with the disease are detected early after birth, which increases the rate of successful treatment.

Specific treatments for membrane disease are active surfactant supplements in high-risk cases. The in dinhation at the time of the child's manifestations of respiratory failure is thought to be late. Surfactant used on the market is derived from natural or synthetic, inttural use to two lungs. Surfactant use can cause many side effects such as bradycardia or apnea.

When respiratory failure occurs, the child should be immediately assisted by oxygen breathing through various instruments. Mechanical ventilators are in order when respiratory failure becomes severe that does not respond to oxygen breathing. Alkaline acid balance disorders need to be adjusted to maintain blood pH at 7.25.

Other supportive treatments include temperature stability, adequate energy supplements and nutrients, electrolye balance also need to be maintained.

The pediatrics department at Share99 International Health Hub is the address to receive and examine the diseases that babies as well as young children are susceptible to: viral fever, bacterial fever, otitis media, pneumonia in children,… With modern equipment, ile space, minimize the impact as well as the risk of spreading the disease. Along with that is the dedication from experienced doctors to pediatric patients, making the examination no longer a concern of parents.

Customers can directly go to Share99 Health System nationwide for examination or contact the hotline HERE for assistance.

VIEW MORE

  • Surfactant pumps for newborn respiratory failure: Things to keep in mind
  • Respiratory failure caused by diaphragm in premature babies
  • Acute respiratory failure in premature babies: Risks and complications
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About: John Smith

b1ffdb54307529964874ff53a5c5de33?s=90&r=gI am the author of Share99.net. I had been working in Vinmec International General Hospital for over 10 years. I dedicate my passion on every post in this site.

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